By Shinjita Das M.D., Rachel Reynolds M.D. (auth.), Joshua Zeichner (eds.)
Dermatologists ordinarily deal with zits, which impacts forty to 50 million americans every year and approximately eighty five percentage of all sufferers sooner or later of their lives. Dermatologists, basic care medical professionals, and pediatricians see those sufferers each day in perform. it is very important deal with the outside successfully not just to minimize the danger of actual scarring, but in addition to handle the detrimental psychosocial effect this sickness incorporates. bettering the surface can increase self-confidence, interpersonal relationships, and function in class or at paintings.
Acneiform Eruptions in Dermatology is a realistic, full-color advisor to the differential prognosis of zits vulgaris and the therapy of acne-like stipulations. it's prepared into sections by means of subtype of (e.g., infections, genetic syndromes, medication-caused) and incorporates a portion of variations of pimples which may be misdiagnosed. in the sections, person chapters talk about every one variation of the and start with a invaluable bulleted precis of its defining scientific gains. vast colour scientific photos seem in the course of the book.
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Additional info for Acneiform Eruptions in Dermatology: A Differential Diagnosis
The melanocortin 5 receptor is expressed in human sebaceous glands and rat preputial cells. J Invest Dermatol. 2000;115(4):614–9. 42. Zouboulis CC, Seltmann H, Hiroi N, Chen W, Young M, Oeff M, Scherbaum WA, Orfanos CE, McCann SM, Bornstein SR. Corticotropin-releasing hormone: an autocrine hormone that promotes lipogenesis in human sebocytes. Proc Natl Acad Sci U S A. 2002;99(10):7148–53. 43. Ganceviciene R, Graziene V, Fimmel S, Zouboulis CC. Involvement of the corticotropinreleasing hormone system in the pathogenesis of acne vulgaris.
Reynolds peripheral blood mononuclear cells and normal human keratinocytes. J Dermatol Sci. 2005;38(1):47–55. Nagy I, Pivarcsi A, Koreck A, Széll M, Urbán E, Kemény L. Distinct strains of Propionibacterium acnes induce selective human beta-defensin-2 and interleukin-8 expression in human keratinocytes through toll-like receptors. J Invest Dermatol. 2005;124(5):931–8. Nagy I, Pivarcsi A, Kis K, Koreck A, Bodai L, McDowell A, Seltmann H, Patrick S, Zouboulis CC, Kemény L. Propionibacterium acnes and lipopolysaccharide induce the expression of antimicrobial peptides and proinflammatory cytokines/chemokines in human sebocytes.
Acnes organisms present on the skin, leading to a reduction in inflammatory pathways and consequently a decrease in the number of inflammatory and noninflammatory acne lesions [24–26]. Several concerns have developed regarding the use of topical antibiotics, specifically surrounding the emergence of resistant strains of P. acnes. Rates of erythromycin and clindamycin resistant P. acnes have increased to 40 % globally, leading to a decrease in treatment efficacy of topical antibiotics [27–29]. Although resistance has emerged to both erythromycin and clindamycin, the clinical efficacy of clindamycin has been preserved relative to erythromycin .